Side effects Oxandrolone



women administered oxandrolone may experience virilization, irreversible development of masculine features such voice deepening, hirsutism, menstruation abnormalities, male-pattern hair loss, , clitoral enlargement. oxandrolone may disrupt growth in children, reducing adult height. because of these side effects, doses given women , children minimized , people monitored virilization , growth abnormalities. other androgens, oxandrolone can cause or worsen acne , priapism (unwanted or prolonged erections). oxandrolone can reduce males fertility, side effect common among androgens. in attempt compensate exogenous increase in androgens, body may reduce testosterone production via testicular atrophy , inhibition of gonadotropic activity.


unlike aas, oxandrolone not cause gynecomastia because not aromatized estrogenic metabolites. however, although no reports of gynecomastia made in spite of widespread use, oxandrolone reported in publication in 1991 have been associated 33 cases of gynecomastia in adolescent boys treated short stature. gynecomastia developed during oxandrolone therapy in 19 of boys , after therapy completed in 14 of boys, , 10 of boys had transient gynecomastia, while 23 had persistent gynecomastia necessitated mastectomy. though transient gynecomastia natural , common occurrence in pubertal boys, gynecomastia associated oxandrolone of late/delayed onset , persistent in high percentage of cases. such, researchers stated, although oxandrolone cannot implicated stimulatory [in] gynecomastia , possible relationship should considered in clinicians using oxandrolone in adolescents growth stimulation.


uniquely among 17α-alkylated aas, oxandrolone shows little no hepatotoxicity, @ high doses. no cases of severe hepatotoxicity have been singularly attributed oxandrolone. however, elevated liver enzymes have been observed in people, particularly high doses and/or prolonged treatment, although return normal ranges following discontinuation. in case, oxandrolone may among safest 17α-alkylated aas in terms of hepatotoxicity.








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